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ERS promotes Mcl-1 downregulation through the PERK/ATF4/CHOP axis while activating the calpain/caspase-12 pathway, forming synergistic crosstalk with mitochondrial apoptosis ( via p21 upregulation and CDK suppression) exacerbate genomic instability and proliferation inhibition (Zhang H

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T., Green, M., Roche, W., Prado-Cabrero, A., Mulcahy, R., & Nolan, J

GLUT1DS patients often show a less severe overall clinical picture than other patients with DRE undergoing KDTs, who are often challenged by partial access to testing or are extremely far from the standard patients for whom existing questionnaires and evaluation tools have been validated

The compound's selectivity profile minimizes concerns about off-target effects, but investigators should consider potential interactions with other NAD+-modulating interventions or compounds that affect methylation metabolism